Selected publications:

1. Rydström and Wick, M.J. (2010). Salmonella inhibits monocyte differentiation into CD11chiMHC-IIhicells in a MyD88-dependent fashion. J. Leukoc. Biol. 87:823-832.
2. Rydström and Wick, M.J. (2009). Monocyte and neutrophil recruitment to intestinal lymphoid tissues during oral Salmonella infection is driven by MyD88-derived chemokines. Eur. J. Immunol. 39:3019-3030.
3. Sundquist, M. and Wick, M.J. (2009) Salmonella-induced death of CD8α+ dendritic cells but not CD11cintCD11b+ inflammatory cells in vivo via MyD88 and TNFR1. J. Leukoc. Biol. 85:225-234.
4. Tam, M. and Wick, M.J. (2009). MyD88 and IFN-αβ are differentially required for dendritic cell maturation but dispensable for development of protective memory againstListeria. Immunology 128:429-438.
5. Tam, M.A.,Sundquist, M., and Wick, M.J. (2008). MyD88 and IFN-αβ differentially control maturation of bystander but not Salmonella-associated dendritic cells or CD11cintCD11b+ cells during infection. Cell. Microbiol. 10:1517-1529.
6. Tam, M.A., Rydström, A., Sundquist, M. and Wick, M.J. (2008). Early cellular responses to Salmonella infection: dendritic cells, monocytes, and more.  Immunol. Rev. 225:140-162.
7. Rydström, A. and Wick, M.J. (2007). Monocyte recruitment, activation and function in the gut-associated lymphoid tissue during oral Salmonella infection. J. Immunol.178:5789-5801.
8. Tam, M.A. and Wick, M.J. (2006).  Differential expansion, activation and effector functions of conventional and plasmacytoid dendritic cells in mouse tissues transiently infected with Listeria monocytogenes. Cell. Microbiol. 8:1172:1187.
9. Sundquist, M. and Wick, M.J. (2005) TNF-dependent and –independent maturation of dendritic cells and recruited CD11cintCD11b+ cells during oral Salmonella infection.  J. Immunol. 175:3287-3298.